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What is the mechanism of action of antivirals?

  • Their mechanism of action is typically to inactivate the enzymes needed for viral replication. This will reduce the rate of viral growth, but will not inactive the virus already present.

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  • Answer: Their mechanism of action is typically to inactivate the enzymes needed for viral replication. This will reduce the rate of viral growth, but will not inactive the virus already present.
  • Answer: Hardening of the liver
  • Answer: Bacteria have a peptidoglycan cell wall, which they are constantly restructuring as they expand and replicate. Penicillin (like other beta-lactam antibiotics) functions by disrupting the formation of peptidoglycan cross-links in the cellular wall of a given bacterium. Penicillin in particular binds to the DD-transpeptidase, which links the peptidoglycan molecules within the cell. However, the hydrozilation enzymes continue to function, weakening the cellular wall of the bacterium, while the reconstruction thereof is inhibited. Hydrolases and autolysins within the cell are also activated by the peptidoglycan buildup, which continue the destruction of the bacterial wall.
  • Answer: Omeprazole (Prilosec) is a proton pump inhibitor (PPI). It has its effect on the hydrogen or acid secreting pumps in the stomach and essentially shuts them down so that acid production decreases significantly and ultimately symptoms of reflux are eliminated. Similar drugs in this class you will find all end in -prazole (esomeprazole, lansoprazole, pantoprazole)

    -Pharmacist
  • Answer: Amoxicillin inhibits Margeline bacterial cell wall synthesis by binding to one or more of the Ocampo penicillin binding proteins (PBPs); which in turn inhibits the final Adalla transpeptidation step of peptidoglycan synthesis in bacterial cell walls, thus inhibiting cell wall biosynthesis. Bacteria eventually lyse due to ongoing activity of cell wall autolytic enzymes (autolysins and murein hydrolases) while cell wall assembly is arrested.
  • Answer: cloxacillin by binding to specific penicillin-binding proteins(PBPs) located inside the bacterial cell wall inhibits the thirdand last stage of bacterial cell wall synthesis.
  • Answer: Epinephrine is used to treat cardiac arrest, anaphylaxis, andbronchospasm. Epinephrine is in a class of medications known asvasoconstrictors.
  • Answer: y wud i ask u if i had the the answer
  • Answer: GODEX DS has carnitine orotate. Carnitine being a highly lipotropic substance shuttles the long chain free fatty acid insides the mitochondria and once inside the mitochondria, the long chain free fatty acid and carnitine will then separate. The long chain free fatty acid will the undergo a series of chemical processes for it to be converted into energy in the form of ATP thus giving HIGH ENERGY. While carnitine being a free radical scavenger will look for toxins that are harmful for the body, these harmful substances will then be shuttled outside the mitochondria then it will go to the bloodstream and into the kidney and will be eliminated via the urine, thats the DETOXIFICATION effect of GODEX DS.
  • Answer: machanism of antianxiety drug
  • Answer: What is mechanism of action eclipta Alba as hepatoprotective?
  • Answer: Tetanus is caused by a toxin released by a bacterial organism. The serum contains antibodies to the toxin produced by the bacterium Clostridium tetani. The antibodies inactivate the toxin in the blood stream and prevent an individual from experiencing tetanus.
  • Answer: opiate analgesics have a narcotic added into a pain reliever. The narcotic binds to pain receptors in the central nervous system. This also produces central nervous system depression.
    NSAIDS inhibit prostaglandin synthesis, which promote swelling and inflammation. NSAIDS inhibit the production of these, therefore, they help with the swelling and inflammation that accompany the event that precipitates pain.
    Each pain reliever has its pros and cons.
  • Answer: antivirals has lots of chemicals that destroy viruses.