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What causes the systolic sound?

  • the systolic sound is caused by ventricular contraction

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  • Answer: the systolic sound is caused by ventricular contraction
  • Answer: when pressure in the sphygmomanometer falls below systolic pressure, blood can again enter the artery. At first, blood enters only at peak systolic pressure, and the sound of blood pulsing through the artery becomes audible through the stethoscope. As the pressure falls further, the sound changes because the vessel is remaining open for longer periods. When the cuff pressure falls below diastolic pressure, blood flow becomes continuous, and the sound of the pulse becomes muffled or disappears completely.
  • Answer: There will be no systolic sound if blood is flowing freely. Thisoccurs when there is no measurable constriction of an artery.
  • Answer: It would depend on whether or not you had this reading on a fairlyregular basis. The systolic number of 165 is higher than normal andshould be evaluated by a doctor.
  • Answer: They systolic reading is the top number of your blood pressure when recorded. What it means it the force of blood on the arterial walls during cardiac contraction.

    The bottom number is the diastolic number. This is the force of blood on the arterial walls during the brief relaxation phase of the heart.
  • Answer: The systolic pressure is measured at the first Karotkoff sound (stage I), it is the first tapping noise that you hear with a stethoscope when taking a manual blood pressure.
  • Answer: It means that the presuure exerted upon contraction of the heart is high too. You can end up with heart problems,
  • Answer: Narrowed arteries
    Kidney Disease
  • Answer: SYSTOLIC: Relating to a systole or happening during a systole.
    But what is a systole?
    It is the contraction of the chambers of the heart(especially the ventricles) to drive blood into the aorta and pulmonary artery.

    DIASTOLIC: Relating to a diastole or happening during a diastole.
    But what is a diastole ?
    The widening of the chambers of the heart between two contractions when the chambers fill with blood is called a diastole.
  • Answer: If you mean what causes the systolic to be high while the diastolic is normal or low the answer is hardening of the arteries. Normally as the pulse of blood comes through the artery the walls stretch to accommodate the wave of blood. As the arteries get stiffer instead of stretching they just cause the pressure to go up.

    If yo are asking what causes high blood pressure then the answer is that we usually do not know. Age, salt intake, weight and lack of activity all contribute. Things like pain , anxiety and stimulants cal also make it go up. Occasionally there is a diagnosable and treatable medical condition that can be reversed but this is the exception.
  • Answer: Systolic, the top and high number, is the most important factor in blood pressure readings. It determines whether an individual has hypertension, or high blood pressure. Systolic is the maximum force exerted by the heart on blood vessels while diastolic is the resting phase of the heart between beats. Before the new millennium it was believed that diastolic was more important.
  • Answer: The normal systolic should be below 120 and the normal diastolicshould be below 80. The systolic is the top number while thediastolic is the bottom number.
  • Answer: Both are, but diastolic is bit more important.
    If your systolic is above 140mm - and Diastolic is up at over 90mm.
    Please see a doctor to cure your high blood pressure sign Asap.
  • Answer: Systolic blood pressure (the top number) tells you how strong the pressure is when your heart beats. A higher top number is usually more cause for concern that the lower number.

    Diastolic blood pressure (the bottom number) tells you how strong the pressure is when the heart is not beating. What this generally tells you is how elastic and resilient your system of vessels are. With age, this number gets lower- particularly after middle age.




    http://www.mayoclinic.org/medical-edge-newspaper-2006/may-29a.html